Search results for "Genetic Diversity and Evolution"

showing 10 items of 12 documents

Effect of Host Species on Topography of the Fitness Landscape for a Plant RNA Virus

2016

[EN] Adaptive fitness landscapes are a fundamental concept in evolutionary biology that relate the genotype of individuals with their fitness. At the end, the evolutionary fate of evolving populations depends on the topography of the landscape, that is, the number of accessible mutational pathways and of possible fitness peaks (i.e, adaptive solutions). For long time, fitness landscapes were only theoretical constructions due to a lack of precise information on the mapping between genotypes and phenotypes. In recent years, however, efforts have been devoted to characterize the properties of empirical fitness landscapes for individual proteins or for microbes adapting to artificial environme…

0301 basic medicine2. Zero hungerbiologyFitness landscapeEcologyHost (biology)ImmunologyRNA virusbiology.organism_classificationMicrobiology03 medical and health sciences030104 developmental biologyGenetic Diversity and EvolutionVirologyInsect ScienceRegional scienceEuropean commissionChristian ministryadaptive fitness landscapes ; experimental evolution ; virus evolutionJournal of Virology
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Frequent coinfection of cells explains functional in vivo complementation between cytomegalovirus variants in the multiply infected host.

2005

In contrast to many other virus infections, primary cytomegalovirus (CMV) infection does not fully protect against reinfection. Accordingly, clinical data have revealed a coexistence of multiple human CMV variants/ strains in individual patients. Notably, the phenomenon of multiple infection was found to correlate with increased virus load and severity of CMV disease. Although of obvious medical relevance, the mechanism underlying this correlation is unknown. A weak immune response in an individual could be responsible for a more severe disease and for multiple infections. Alternatively, synergistic contributions of variants that differ in their biological properties can lead to qualitative…

Human cytomegalovirusMuromegalovirusImmunologyPopulationGreen Fluorescent ProteinsBiologymedicine.disease_causeMicrobiologyHerpesviridaeVirusMiceViral ProteinsBetaherpesvirinaeVirologymedicineAnimalseducationLungeducation.field_of_studyMice Inbred BALB CIntegrasesVirulenceGenetic VariationInborn immunodeficiencyCytomegalovirusmedicine.diseasebiology.organism_classificationVirologyGenetic Diversity and EvolutionInsect ScienceImmunologyCytomegalovirus InfectionsCoinfectionNIH 3T3 CellsFemaleSpleenJournal of virology
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Relationship between within-host fitness and virulence in the vesicular stomatitis virus: correlation with partial decoupling.

2012

ABSTRACT Given the parasitic nature of viruses, it is sometimes assumed that rates of viral replication and dissemination within hosts (within-host fitness) correlate with virulence. However, there is currently little empirical evidence supporting this principle. To test this, we quantified the fitness and virulence of 21 single- or double-nucleotide mutants of the vesicular stomatitis virus in baby hamster kidney cells (BHK-21). We found that, overall, these two traits correlated positively, but significant outliers were identified. Particularly, a single mutation in the conserved C terminus of the N nucleocapsid (U1323A) had a strongly deleterious fitness effect but did not alter or even …

ImmunologyMutantVirulenceApoptosisBiologymedicine.disease_causeVirus ReplicationMicrobiologyVesicular stomatitis Indiana virusCell Line03 medical and health sciencesVesicular StomatitisMiceVirologyCricetinaemedicineBaby hamster kidney cellAnimals030304 developmental biologyGlycoproteinsGenetics0303 health sciencesMutationMice Inbred BALB CVirulence030302 biochemistry & molecular biologyCell MembraneBrainNucleocapsid Proteinsbiology.organism_classification3. Good healthProtein Structure TertiaryViral replicationGenetic Diversity and EvolutionVesicular stomatitis virusInsect ScienceMutationFemaleNeuron deathVesicular StomatitisJournal of virology
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Nucleoside Analogue Mutagenesis of a Single-Stranded DNA Virus: Evolution and Resistance

2012

ABSTRACT It has been well established that chemical mutagenesis has adverse fitness effects in RNA viruses, often leading to population extinction. This is mainly a consequence of the high RNA virus spontaneous mutation rates, which situate them close to the extinction threshold. Single-stranded DNA viruses are the fastest-mutating DNA-based systems, with per-nucleotide mutation rates close to those of some RNA viruses, but chemical mutagenesis has been much less studied in this type of viruses. Here, we serially passaged bacteriophage ϕX174 in the presence of the nucleoside analogue 5-fluorouracil (5-FU). We found that 5-FU was unable to trigger population extinction for the range of conce…

Mutation rateGenes ViralImmunologyBiologyMicrobiology03 medical and health scienceschemistry.chemical_compoundTranscription (biology)VirologyDrug Resistance ViralGenePolymerase030304 developmental biologyGenetics0303 health sciences030302 biochemistry & molecular biologyRNARNA virusDNAbiology.organism_classificationVirology3. Good healthGenetic Diversity and EvolutionchemistryInsect ScienceSingle Stranded DNA VirusMutagenesis Site-Directedbiology.proteinFluorouracilDirected Molecular EvolutionBacteriophage phi X 174DNAJournal of Virology
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Delayed lysis confers resistance to the nucleoside analogue 5-fluorouracil and alleviates mutation accumulation in the single-stranded DNA bacterioph…

2014

ABSTRACT Rates of spontaneous mutation determine viral fitness and adaptability. In RNA viruses, treatment with mutagenic nucleoside analogues selects for polymerase variants with increased fidelity, showing that viral mutation rates can be adjusted in response to imposed selective pressures. However, this type of resistance is not possible in viruses that do not encode their own polymerases, such as single-stranded DNA viruses. We previously showed that serial passaging of bacteriophage ϕX174 in the presence of the nucleoside analogue 5-fluorouracil (5-FU) favored substitutions in the lysis protein E (P. Domingo-Calap, M. Pereira-Gomez, and R. Sanjuán, J. Virol. 86: 9640–9646, 2012, doi:10…

Mutation rateImmunologyAdaptation BiologicalMutation MissenseDNA Single-Strandedmedicine.disease_causeMicrobiologyBacteriophage03 medical and health scienceschemistry.chemical_compoundViral ProteinsBacteriolysisMutation RateVirologymedicineBacteriophagesSelection GeneticPolymerase030304 developmental biologyGenetics0303 health sciencesMutationbiology030302 biochemistry & molecular biologyMutagenesisMutation AccumulationResistance mutationbiology.organism_classificationVirologychemistryGenetic Diversity and EvolutionInsect ScienceDNA Viralbiology.proteinMutant ProteinsFluorouracilDNAJournal of virology
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Changes in protein domains outside the catalytic site of the bacteriophage Qβ replicase reduce the mutagenic effect of 5-azacytidine.

2014

ABSTRACT The high genetic heterogeneity and great adaptability of RNA viruses are ultimately caused by the low replication fidelity of their polymerases. However, single amino acid substitutions that modify replication fidelity can evolve in response to mutagenic treatments with nucleoside analogues. Here, we investigated how two independent mutants of the bacteriophage Qβ replicase (Thr210Ala and Tyr410His) reduce sensitivity to the nucleoside analogue 5-azacytidine (AZC). Despite being located outside the catalytic site, both mutants reduced the mutation frequency in the presence of the drug. However, they did not modify the type of AZC-induced substitutions, which was mediated mainly by …

Mutation rateImmunologyMutantRNA-dependent RNA polymeraseBiologyVirus ReplicationMicrobiologyViral ProteinsVirologyCatalytic DomainmedicineGeneticsAllolevivirusNucleoside analogueQ beta Replicasebiology.organism_classification3. Good healthProtein Structure TertiaryViral replicationBiochemistryAmino Acid SubstitutionGenetic Diversity and EvolutionInsect ScienceAzacitidineQ beta ReplicaseBacteriophage QβNucleosidemedicine.drugMutagensJournal of virology
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Viral Mutation Rates

2010

Accurate estimates of virus mutation rates are important to understand the evolution of the viruses and to combat them. However, methods of estimation are varied and often complex. Here, we critically review over 40 original studies and establish criteria to facilitate comparative analyses. The mutation rates of 23 viruses are presented as substitutions per nucleotide per cell infection (s/n/c) and corrected for selection bias where necessary, using a new statistical method. The resulting rates range from 108 to106 s/n/c for DNA viruses and from 106 to 104 s/n/c for RNA viruses. Similar to what has been shown previously for DNA viruses, there appears to be a negative correlation between mut…

Mutation rateTime FactorsvirusesImmunologyBiologyMicrobiologyVirusEvolution Molecularchemistry.chemical_compoundVirologyAnimalsHumansRNA VirusesNucleotideIndelGenome sizechemistry.chemical_classificationGeneticsModels GeneticDNA VirusesRNAVirologyGenetic Diversity and EvolutionchemistryInsect ScienceMutationVirusesMutation (genetic algorithm)DNA
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Effect of Ribavirin on the Mutation Rate and Spectrum of Hepatitis C Virus In Vivo

2009

ABSTRACTTheir extremely error-prone replication makes RNA viruses targets for lethal mutagenesis. In the case of hepatitis C virus (HCV), the standard treatment includes ribavirin, a base analog with an in vitro mutagenic effect, but the in vivo mode of action of ribavirin remains poorly understood. Here, we test the mutagenic effects of ribavirin plus interferon treatment in vivo using a new method to estimate mutation rates based on the analysis of nonsense mutations. We apply this methodology to a large HCV sequence database containing over 15,000 reverse transcription-PCR molecular clone sequences from 74 patients infected with HCV. We obtained an estimate of the spontaneous mutation ra…

Mutation ratevirusesHepacivirusHepatitis C virusImmunologyNonsense mutationHepacivirusmedicine.disease_causeMicrobiologyViruschemistry.chemical_compoundInterferonVirologyRibavirinmedicineHumansbiologyRibavirinvirus diseasesbiology.organism_classificationVirologyMolecular biologydigestive system diseasesGenetic Diversity and EvolutionchemistryViral replicationCodon NonsenseInsect ScienceMutationmedicine.drugJournal of Virology
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Are Human P[14] Rotavirus Strains the Result of Interspecies Transmissions from Sheep or Other Ungulates That Belong to the Mammalian Order Artiodact…

2009

ABSTRACT A limited number of human G6P[14] rotavirus strains that cause gastroenteritis in humans have been isolated in Europe and Australia. The complete genome sequences were determined for five of these human strains—B10925-97 (isolated in Belgium in 1997), 111/05-27 (Italy, 2005), PA169 (Italy, 1987), MG6 (Australia, 1993), and Hun5 (Hungary, 1997)—and their genetic relatedness to animal rotavirus strains was evaluated by sequencing the complete genome of the sheep rotavirus OVR762 (G8P[14]; Spain, 2002), the guanaco ( Lama guanicoe ) rotavirus strains Arg/Chubut/99 and Arg/Río Negro/98 (G8P[14] and G8P[1], respectively; Argentina, 1999 and 1998), the sable antelope strain RC-18/08 (G6…

RotavirusSequence analysisImmunologyPopulationMolecular Sequence DataReoviridaeSequence HomologyBiologymedicine.disease_causeMicrobiologyDNA sequencingRotavirus InfectionsEvolution MolecularPhylogeneticsVirologyRotavirusGenotypemedicineHumanseducationPhylogenyGeneticseducation.field_of_studyPhylogenetic treeAustraliaInfantSequence Analysis DNAbiology.organism_classificationVirologyGastroenteritisEuropeGenetic Diversity and EvolutionInsect ScienceChild PreschoolRNA Viral
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Heterogeneity and temporal dynamics of evolution of G1 human rotaviruses in a settled population.

2006

ABSTRACT A rotavirus sample collection from 19 consecutive years was used to investigate the heterogeneity and the dynamics of evolution of G1 rotavirus strains in a geographically defined population. Phylogenetic analysis of the VP7 gene sequences of G1P[8] human rotavirus strains showed the circulation of a heterogeneous population comprising three lineages and seven sublineages. Increases in the circulation of G1 rotaviruses were apparently associated with the introduction of novel G1 strains that exhibited multiple amino acid changes in antigenic regions involved in rotavirus neutralization compared to the strains circulating in the previous years. The emergence and/or introduction of G…

RotavirusSerotypeSettore MED/07 - Microbiologia E Microbiologia ClinicaGenes ViralSettore MED/17 - Malattie InfettiveSequence analysisvirusesLineage (evolution)Molecular Sequence DataImmunologyPopulationViral Nonstructural ProteinsBiologymedicine.disease_causeMicrobiologyRotavirus InfectionsEvolution Molecularfluids and secretionsPhylogeneticsVirologyRotavirusmedicineHumansAmino Acid SequenceLongitudinal StudiesSerotypingeducationAntigens ViralPhylogenyGeneticseducation.field_of_studySequence Homology Amino AcidPhylogenetic treeInfantRNA-Binding Proteinsvirus diseasesVirologyItalyGenetic Diversity and Evolutionrotavirus G1Child PreschoolInsect ScienceCapsid ProteinsSample collection
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